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Fig. 8 | Phytopathology Research

Fig. 8

From: The phytopathogen Xanthomonas campestris scavenges hydroxycinnamic acids in planta via the hca cluster to increase virulence on its host plant

Fig. 8

A schematic working model for hca induction in Xcc. a In the absence of HCAs or at the epiphytic stage of infection, the hca gene cluster is transcribed at a low level. HcaR binds to site 1 of the hca promoter Phca to inhibit hca transcription. b In the presence of HCAs or during the in planta stage, HCAs are transported into the cytosol and bind to an uncharacterized HcaX, the resultant HCA/HcaX complex binds to site 2 of Phca to increase hca transcription. The increased levels of HcaL catalyze the conversion of HCAs into HCA-CoAs, which further bind to HcaR. The HCA-CoA/HcaR complex then releases from the site 1 of Phca, further increasing the transcription level of hca. This is a self-reinforcing process which eventually leads to very high levels of expression of the hca gene cluster

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Phytopathology Research

ISSN: 2524-4167