Fig. 8From: An increase in the number of peroxisomes is coupled to the initial infection stage and stress response of Botrytis cinereaWorking model of fatty acid oxidation in peroxisomes during the initial stage of B. cinerea infection. The contents in brackets represent the symbols according to the KEGG results. ABCD, ATP-binding cassette, subfamily D; ACAA, Acetyl-CoA acyltransferase; ACOX, Acyl-CoA oxidase; ACSL, Long-chain acyl-CoA synthetase; AMACR, α-methylacyl-CoA racemase; CAT, Catalase; CRAT, Carnitine O-acetyltransferase; ECH, δ(3,5)-δ(2,4)-dienoyl-CoA isomerase; HPCL, 2-hydroxyacyl-CoA lyase; NUDT, Diphosphatase; PDCR, 2,4-dienoyl-CoA reductase; PECI, δ(3)-δ(2)-enoyl-CoA isomerase; pex, Peroxin; PMP and PXMP, Peroxisomal membrane protein; PTE, Acyl-CoA thioesterase; SCPX, Sterol carrier protein; SOD, Superoxide dismutaseBack to article page